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In highly differentiated human CD4 T cells (the CD27CD28 subset), AMPK activation under metabolic stress or DNA damage recruits TAB1 to induce p38 autophosphorylation, leading to telomerase suppression and proliferative arrest 44

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The peptide's actin-sequestering activity enables the rapid cytoskeletal reorganization required for keratinocyte migration and fibroblast movement into wound sites, with studies showing 2-3 fold increases in migration rates and improved directional persistence compared to untreated controls

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