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Therefore, in the periphery, extracellular GSH reflects the antioxidant capacity of the liver, and the liver, due to its function in metabolizing xenobiotics and endogenous molecules, has high antioxidant capacity [16]

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Moreover, NO production in the vascular endothelium of the penis has been found to be decreased in vasculogenic ED, such as in the case of ED caused by atherosclerosis and diabetes [16, 17, 19], and a reduction of circulating l-ARG levels has been detected in patents with vasculogenic ED [20], further confirming that l-ARG supplementation might be beneficial in the treatment of vasculogenic ED