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We next repeated this experimental paradigm in Gipr hypo-KO and Gipr hypo-GFP mice, combining the initiation of HFD with a combination of LIRA (or VEH) alone or LIRA (or VEH) plus GIPR antagonist peptide (GIPR-antag)

These results suggest that NRF2 is a necessary component of melatonin signaling pathways in ILC2s, linking the canonical circadian hormone to cellular redox machinery