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Description

Both A deposition, misfolded, and hyperphosphorylated tau protein are shown to disrupt Ca 2+ homeostasis in mitochondria, contributing to AD progression by promoting superoxide generation, metabolic dysfunction, and neuronal cell death (Calvo-Rodriguez and Bacskai, 2021)

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Unfortunately, this leaves few ways to tell exactly how much glutathione makes it into the cells at all

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Evaluation of glutathione S-transferase P1 (GSTP1) Ile105Val polymorphism and susceptibility to type 2 diabetes mellitus, a meta-analysis

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