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Cardiomyocyte survival promotion through ILK/PI3K/Akt anti-apoptotic signalling, cardiac progenitor cell activation and migration toward the ischaemic injury border zone, angiogenesis stimulation in the peri-infarct region to restore oxygen delivery to at-risk myocardium, and anti-fibrotic effects that attenuate the non-functional scar tissue formation that impairs cardiac function following myocardial infarction