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2 Although alcohol may directly activate stellate cells to produce collagen independently of inflammation and necrosis, 53 the key mediator in alcohol-induced liver disease is acetaldehyde, the product of alcohol metabolism by the enzyme alcohol dehydrogenase

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However, administration of GH and subsequent GH-mediated effects were found to be dose dependent, as high concentrations given to GH-deprived canines caused atrophy of testes and accessory organs, thinning of prostatic epithelium and a reduction in LH and testosterone levels (75)