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Importantly, the GAP activity of TBC1D9B towards ARL8B was comparable to its ability to stimulate GTP hydrolysis by its known substrate RAB11A (Supplementary Fig

Either way, the practical implication is similar, and it is arguably the most consequential consideration for anyone planning future work: on the brain-access hypothesis, novel dual GLP-1/GIP agonists engineered specifically for brain entry would be more rational next candidates to test than repurposed diabetes drugs chosen for market availability