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We suggest glutathione is one such endogenous source, as further impairing glutamate liberation from the cycle diminishes synaptic activity induced by MeAIB, while increasing glutamate availability can rescues the impairment by MeAIB

Racemization: Succinimide intermediates formed during asparagine deamidation are highly prone to racemization and convert to D-asparagine residues (41,42)