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Differential Functional Expression of Human Myocardial G Protein Receptor Kinases in Left Ventricular Cardiac Diseases

These alterations are theorized to contribute to an extended half-life and heightened biological activity compared to endogenous IGF-1

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For example, mutants in the fog-1 gene, which encodes a CPEB-related RNA-binding protein, also enhance the GSC defect of fbf-1 fbf-2 double mutants, but that enhancement is coupled to a reversal in germline sex and the mechanism remains a mystery (THOMPSON et al