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Given the complexity of mitochondrial dysfunction, future strategies may emphasize adjunctive combination therapiesa personalized mito-cocktail combining NAD+ precursors to boost biogenesis, mitochondria-targeted antioxidants to reduce stress, and metabolic modulators to optimize fuel utilization, all tailored to individual mitochondrial phenotype

Concluding remarks Numerous animal studies demonstrate IGF-I as a powerful growth factor for muscle repair

Depletion of GSH and overproduction of ROS in the lens of major intrinsic protein (MIP) mutant mice result in increased expression of Atf3 and Ddit3 63