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[31] Side effects of the antithyroid medications include a potentially fatal reduction in the level of white blood cells

After knockdown the GPX4 gene in epithelial cells, the researchers found that NO inhibits ferroptosis in epithelial cells even when GPX4 is deficient

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In a model of LLC CAC, tumor-derived parathyroid-hormone-related protein (PTHrP) could promote an energy wasting state by reprogramming a thermogenic state in adipose tissue, which involved the upregulation of lipolytic enzymes (221)