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Relevant studies demonstrate that the gut microbiota can regulate the polarization of CD4+ T cells during pulmonary fungal infection, thereby influencing lung immune defense mechanisms

Taken together, these results have demonstrated that BPC-157 induces proliferation, migration, and tube formation of endothelial cells, wherein the ERK1/2 signaling pathway plays a promoting role

Some experimental models indicate that GHK-Cu may regulate fibrosis-related gene pathways, suggesting a possible role in conditions involving abnormal tissue scarring

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