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Experimentally, periodically interrupted or long-lasting administration of an A 2A R antagonist such as SCH58261 combined with L-DOPA may restore normal motor function in PD animal models [23]

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In the obese state, EAT expansion induces a pro-inflammatory and pro-oxidative cardiac microenvironment, promotes ectopic infiltration of cardiomyocytes, and thereby contributes to the development of insulin resistance, endothelial dysfunction, mitochondrial dysfunction, and myocardial fibrosis (63)

10.1371/journal.pone.0106890 5 Al-ZamelN.Al-SabahS.LuqmaniY.AdiL.ChackoS.SchneiderT