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UA administration to mice before the onset of clinical EAE may protect the integrity of the BBB from changes in ONOO -induced permeability, thus minimizing the infiltration of inflammatory cells and other pathogenic cells and factors and the resulting pathology [193](Fig

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This GSH conjugation is spontaneous or enzymatic via GSH-S-transferases [21,22]

Cell Rep 12(10):16311643 Tapias V, Hu X, Luk KC, Sanders LH, Lee VM, Greenamyre JT (2017) Synthetic alpha-synuclein fibrils cause mitochondrial impairment and selective dopamine neurodegeneration in part via iNOS-mediated nitric oxide production