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Remarkably, astrocytes can exert opposing effects on different synaptic inputs depending on the adenosine receptor subtype
Such alterations were also associated with inhibition of cytochrome c oxidase (CcOX) activity and microtubule network disassembly, which are considered hallmarks of nitric oxide (NO) toxicity, indicating that NO, rather than the depletion of GSH per se, is the primary mediator of cell damage
It is also explored for its role in maintaining strong immune response through support of white blood cell activity
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