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In the APP (Swe)/[PS1(L166P)] transgenic (Tg) model of AD, germ-free status reduced cerebral A load compared to conventionally raised APP rodents (Ling et al., 2020)

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Canagliflozin significantly inhibits inflammatory factor expression, including cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase, exerting cardioprotective effects by regulating inflammation and ferroptosis through the AMP-activated protein kinase (AMPK) pathway (83)

That is a meaningful shift for the clinical compounding ecosystem but it is not the same as FDA approval of a finished drug product