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Acknowledgements This work was supported by the National Key Research and Development Project of China (2023YFC3402100), the National Natural Science Foundation of China (81821002 and 82130082, 82341004), 135 project for disciplines of excellence, West China Hospital, Sichuan University (ZYGD22007)

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Histone deacetylase (HDAC) inhibitorssuch as vorinostat and valproic acidhave been shown to induce CSC differentiation and impair tumor-initiating capacity in preclinical models

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