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There is a convincing possibility for treatments intended to markedly enhance the intracellular ROS to execute the cancer cells by diminishing their antioxidant capabilities
Although preclinical studies in colon cancer models show that IDO1 inhibition can enhance T cell infiltration when combined with antiCTLA-4 therapy (125, 126), clinical trials of IDO1 monotherapy have demonstrated limited efficacy (127), reflecting tumor-specific metabolic adaptations and compensatory mechanisms
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