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The genetic test reveals predispositions in cellular antioxidant defense pathways, including variants affecting glutathione metabolism

Gln depletion therapy can inhibit Gln metabolism in tumor cells, but in colon cancer, it has been observed that Gln depletion promotes the expression of the aspartate/glutamate transporter SLC1A3 in tumor cells, increasing the intracellular glutamate concentration and promotes Gln synthesis

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In addition, higher levels of ROS have been shown to activate death receptors via downregulation of the FLIPa protein that regulates apoptosis and functions as a key link between cell death and survival pathways (Wang et al