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Treatment with Dihexa has been shown to reliably activate c-Met activity through HGF-dependent interactions (Benoist et al., Attenuation of aminoglycoside-induced hair cell death can result from two mechanistic categories: blockage of aminogly-coside uptake by hair cells or modulation of intracellular signaling pathways

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What matters is keeping it separated from a claim that the medication modifies the autoimmune disease itself

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