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During TB infection, HDT modulates host cell functions, enhances protective immune responses, and improves the mycobacterial killing activities of host immune cells, working to eradicate or limit TB infection [4, 5]

Findings from siRNA silencing and plasmid amplification of transferrin receptor (TFRC) suggested TFRC is crucial for TMPs ability to mitigate ferroptosis and decrease LDH release, Fe 2+ buildup, and intracellular ROS

Unlike oral supplements that need to be digested and absorbed through the gastrointestinal tract, IV therapy ensures maximum bioavailability and fast results

The collapse of both astrocyte and microglia into pro-inflammatory states due to depletion of GSH, leading to sustained neuroinflammation and oxidative stress, is observed in many neurodegenerative diseases (Figure 2)