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A recent study has examined the effect of cobalamin supplementation on the outcomes of treatment for feline patients with severe cobalamin deficiency and histories suggesting chronic gastrointestinal disease.5 In this study, serum concentrations of methylmalonic acid normalized following parenteral cobalamin supplementation, indicating that cobalamin deficiency was the cause of the high methylmalonic acid in serum

We further investigated the extent of phosphorylation of CREB, the major downstream effector of BDNF [51, 52], as marker of BDNF signalling

Important altered metabolites, including Orn, Pip, and C2, were screened for distinguishing gout patients from AHU patients by random forest analysis

Defective or insufficient mitochondrial function at the cellular level is thought to impact whole-body metabolic homeostasis