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Vitamin B12 is crucial for several physiological processes, including: Red blood cell formation deficiency leads to megaloblastic anaemia, characterised by large, immature red blood cells Neurological function B12 is required for myelin synthesis and nerve health DNA synthesis essential for cell division and growth Without adequate B12, patients may develop symptoms such as fatigue, pallor, shortness of breath, glossitis (sore tongue), paraesthesia (pins and needles), and in severe cases, neurological complications including peripheral neuropathy, ataxia, and cognitive impairment

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Accordingly, 24 proteins involved in translation were 1.52.8-fold upregulated under SARS-CoV-2 infection, including the translation factor EIF2B1, ribosomal proteins (RPL26, MRPS30, RRP8, PDCD11, and MRPL4), RNA helicases (DDX55 and DDX56), RNAses (POP1 and XRN1) and other regulatory factors, such as phosphatases (PPP1CC, PPP1CA, and PPP2R5A) (Supplementary Table 3 and Figure 5A)