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doi:10.1073/pnas.84.10.3434

Furthermore, Protein-sol revealed that our engineered fusion constructs were more soluble than the soluble E

PPAR/ might also be responsible for the reduced GLP-1 production due to NAD + depletion, as it has been demonstrated to transcriptionally regulate proglucagon expression in enteroendocrine L cells by stimulating the -catenin/TCF7L2 pathway [138]

In contrast, our analysis provided a more in-depth exploration of this relationship and further incorporated drug-target MR analyses to evaluate the potential therapeutic implications of GLP-1RAs, a class of antidiabetic drugs, for individuals with SA