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Unlike GLP-1 agonists, which primarily act by delaying gastric emptying and enhancing insulin secretion, BRP operates via the following mechanisms: Hypothalamic activation : BRP triggers the expression of the FOS gene, a key marker of neuronal activation associated with appetite suppression, in the hypothalamus

[32] It also shows up in research on muscle recovery, with supplementation linked to lower markers of muscle damage and soreness following hard exercise

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None of these concerns invalidates the drug