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It works by selectively binding to the ghrelin receptor (GHS-R1a) a key trigger in the GH release cascade

This first-pass mechanism of periportal toxicity for selected compounds, including hepatotoxicants that primarily affect centrilobular hepatocytes, could result from deficient cytoprotective capacity secondary to depletion of phase II conjugating enzymes in addition to being a consequence of a higher exposure gradient to these upstream hepatocytes

Collectively, these findings support the hypothesis that retrograde transport may mediate the retrieval of VSRs from MVBs