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Preventive effects of exenatide on endothelial dysfunction induced by ischemia-reperfusion injury via KATP channels
Comparison of inactive GLP-1R and GCGR Comparison of the inactive GLP-1RFab7F38 structure with the previous inactive GCGRmAb1 structure 14 reveals a remarkable similarity of the inner halves of the TMD, which is consistent with the high sequence identity and common Gs coupling of the two receptors
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Enrichment analysis was carried out on these DEGs to identify key biological pathways altered in eWAT following HFD induction and Retatrutide intervention
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