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Interestingly, it also stimulates insulin secretion from -cells, but with much lower potency than GLP-1 with the mechanism potentially due to the weak binding of glucagon at GLP-1 receptor site [11]

doi: 10.2337/dc12-2454 72 RosenstockJHanefeldMShamannaPMinKWBokaGMiossecPet al

Richards reports serving on the advisory board for Rhythm Pharmaceuticals and is a speaker for Novo Nordisk, Eli Lilly, and Rhythm Pharmaceuticals

GLP-1 receptor agonism is well established to slow gastric emptying, which can increase intra-gastric pressure and potentially promote acid reflux in susceptible individuals