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EC 1.14.13.39), suggesting that chronic glial activation can cause oxidative stress in the brain, similarly to that seen in neurodegenerative processes like AD and Parkinson [10, 39, 45]

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Although GSH is not alone responsible for these protective mechanisms, the hepatic production of GSH seems to be essential to survival, with knockout mice dying within months secondary to inflammation and hepatic steatosis resulting in liver failure [21]

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