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The mitochondrial release of apoptosis-inducing factor (AIF) and of endonuclease G (EndoG), which is extremely specific for myonuclei, occur, which are responsible for DNA fragmentation and chromatin condensation, as well as the collapse of the nuclear envelope ( Fig

Over time, your body might adjust to the medication, which can affect its effectiveness

However, current evidence suggests that the disorder arises from a complex interplay of genetic predisposition, altered monoamine neurotransmission, and structural brain abnormalities, which together increase susceptibility

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