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Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)

Anti-fibrotic and anti-inflammatory signals An apparent paradox in the GHK-Cu data is that the same molecule described as pro-synthetic (it increases collagen) is also described as anti-fibrotic

The combination with GHK-Cu provides both the structural components (collagen, extracellular matrix) and the cellular mobility needed for optimal tissue repair