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In addition to the remaining ACE2 expression, another possible explanation for this heterogeneity in COVID-19 susceptibility could be that that independently of ACE2, the quality of life for people with cystic fibrosis may depend on contributions from non-CFTR modifier or bystander genes 69,70,71,72,73,74,75

However, clinically successful treatment strategies are still lacking and are often inspired by treatment options for diseases with similar clinical presentations, such as ME/CFS

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Cysteine cathepsin inhibitors as anti-Ebola agents