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In Australia, BPC-157 for therapeutic or human use is a Schedule 4 substance and requires a prescription

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Increased expression of the FMO3 gene could be a culprit in atherosclerosis and CVD.8 FMO3 may have adverse effects on blood lipids and glucose independently of TMAO formation, although more research is needed.15 Also, in the liver, trimethyllysine (TML), a modified amino acid, can be converted to GBB and then to carnitine and potentially to TMA and TMAO

This duration-dependent relationship held up specifically in cardiovascular outcome trials, where the odds ratio reached 2.18 (95% CI: 1.36-3.49)