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Vanadium derivative exposure promotes functional alterations of VSMCs and consequent atherosclerosis via ROS/p38/NF-kappaB-mediated IL-6 production

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To further substantiate that inhibiting GJs is an effective means of protecting against APAP-induced hepatotoxicity, we utilized the GJ functional inhibitor 2-aminoethoxydipenyl borate (2APB) (Leytus et al

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