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In preclinical research models, MT-2 has been characterized for its agonism at MC1R (the receptor governing melanocyte eumelanin synthesis) and at MC4R (a central receptor studied in energy-homeostasis and melanocortin-signaling research)
Further, 9-me-BC appears to decrease toxic inflammation in the brain by decreasing inflammatory cytokines which are known to disrupt cognitive function through microglial accumulation
In comparison to the wild type mice, APP/PS1 mice experienced significant synaptic loss as well as a reduction in the number of neuronal cells in the cerebral cortex
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