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Additionally, it supports the activity of Superoxide Dismutase (SOD) , providing a model for studying cellular protection against oxidative stress

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The destructive factors include stomach hydrochloric acid (HCl), mucosal hypoperfusion, free oxygen radicals, ethanol, Helicobacter pylori and excessive ingestion of non-steroidal anti-inflammatory drugs (NSAIDs) that promote the gastric mucosal injury and contribute to gastric ulceration [3]

Quek YLK, Li J, Fujimoto J, Zhang J, Chow CWB, Behrens C, Mao X, Correa A, Song X, Zhang J, Roarty EB, Thornton RL, Coyle MD, Antonoff MB, Kalhor N, Moran C, Weissferdt A, William Jr WN, Swisher SG , Lee JJ, Heymach JV, Wistuba IV, Futreal A, Zhang J