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Product Attributes CAS # : 49557-75-7 Molecular Formula : C14H24N6O4Cu Sequence (AA) : C14H24N6O4 (free peptide), C14H22CuN6O4 (copper complex) Molecular Weight : 340.38 g/mol (peptide), 403.92 g/mol (copper complex) PubChem CID : 71587328 Half-Life : ~1-2 hours Synonyms : Copper Tripeptide-1, Cu-GHK, GHK-Cu Complex, Tripeptide-Copper, Prezatide, Kollaren, Glycyl-Histidyl-Lysine Type : Naturally occurring peptide-metal complex Research Focus : Beauty & Skin, Anti-Aging, Regeneration, Hair Growth Scientific References Expression of glycosaminoglycans and small proteoglycans in wounds: modulation by the tripeptide-copper complex glycyl-L-histidyl-L-lysine-Cu2+ Animal The human tripeptide GHK-Cu in prevention of oxidative stress and inflammatory conditions Review Regenerative and protective actions of the GHK-Cu peptide in the light of the new gene data Review The potential of GHK as an anti-aging peptide Review GHK peptide as a natural modulator of multiple cellular pathways in skin regeneration Review Thermodynamically stable ionic liquid microemulsions pioneer pathways for topical delivery and peptide retention In vitro Exploring the beneficial effects of GHK-Cu on an experimental model of DSS-induced ulcerative colitis in mice Animal The human tri-peptide GHK and tissue remodeling Review The tripeptide-copper complex glycyl-L-histidyl-L-lysine-Cu2+ stimulates matrix metalloproteinase-2 expression by fibroblast cultures In vitro In vivo stimulation of connective tissue accumulation by the tripeptide-copper complex glycyl-L-histidyl-L-lysine-Cu2+ in rat experimental wounds Animal Included In The Box Every product arrives in a premium, custom-designed PEPTIDE.Power box , engineered for convenience, hygiene, and safe storage in your refrigerator
An improved understanding of the relationship between the folate system, UVR environment, and folate-related health outcomes may provide new insight into humankinds continued vulnerability to these diseases
Other studies show that calcium oxide crystals induce mitochondrial dysfunction in macrophages.[ref][ref] High dietary oxalate intake causes some, but not all, people to have statistically significant decreases in mitochondrial metabolic function.[ref] Animal models of high oxalate levels show that there is an increase in mitochondrial reactive oxygen species (ROS) that can lead to oxidative stress if glutathione is inadequate
