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The NRF2-dependent effect was identified through the antioxidative response element in target gene loci, while NRF2-independent effects act through DNA 5hmC modifications via the regulation of the metabolite L2HG

Evidence comparison: GHK-Cu has a substantially larger body of published research, spanning from Pickart's original 1973 isolation through hundreds of subsequent studies

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Species differences Of note, GIPR agonists and GIP-1R agonists alone failed to increase the beating rate in RA or FOC in LA (Neumann et al