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Endothelial IGF- 1R knockdown impairs skeletal muscle strength without causing significant muscle atrophy Skeletal muscle mass plays a critical role in determining the maximal force output, and its reduction can severely impair muscle strength [52,53,54,55]

Comparative metabolic and hepatic effects of GLP-1-based mono-, dual-, and triple-agonist therapies GLP-1 receptor signaling Intestinal L-cells serve as the primary source of GLP-1 synthesis, which operates via G-protein coupled receptors present in pancreatic beta cells, gastrointestinal and central neural systems, cardiac tissues, and kidneys [26]

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CJC-1295 binds to receptors in the pituitary gland to stimulate the release of growth hormones plasma growth hormone (GH) and insulin-like growth factor 1 (IGF-1) [4]