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APP carriers presented an accumulation of naive T cells at the expense of the contraction of highly differentiated memory cells, accompanied by a remodeling of the plasma metabolome that favored a decrease in the NAD + precursor trigonelline 27 , shown to restore memory function in 5xFAD mice 67 , along with an increase in adenosine, reported to mediate non-canonical CD38-dependent immunosuppression 68

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While the rationale for ENaC inhibition has been extensively explored in cystic fibrosis (CF) due to its role in CFTR-dependent secretion, it is critical to distinguish these mechanisms from the pathophysiological environment of non-CF bronchiectasis

Am J Physiol Cell Physiol 287:C565571 Matschinsky FM (1990) Glucokinase as glucose sensor and metabolic signal generator in pancreatic beta-cells and hepatocytes