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In brief, these factors encompass direct toxicity from prolonged thyroid hormones excess, autoimmune-mediated injury, heart failurerelated hepatic necrosis, increased metabolic rate and oxidative stress, and potential hepatotoxicity from antithyroid medications 64,65,66,67

BPC 157 also promoted the internalization of VEGFR2, a critical step for receptor activation

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Oxidative stress was selected as the experimental model because it is widely recognized as a central mechanism in cataract development and can directly disrupt lens epithelial cell homeostasis