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Key Takeaways GHK-Cu is a copper binding tripeptide, not a hormone It modulates extracellular matrix remodeling Its effects are local rather than endocrine Human evidence supports skin and wound applications Systemic anti aging claims exceed available data Sources Pickart L, Thaler MM

LPS challenges induced MD2-TLR4 interaction, phosphorylation of TAK1, MAPKs, TBK1, and IRF3, and NF-B activation, while rBDNF treatment reversed these changes in mouse lung tissues (Supplementary Fig

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Histone lactylation as a driver of metabolic reprogramming and immune evasion