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Studies in multiple model organisms have demonstrated that this age-related NAD+ decline correlates with mitochondrial dysfunction, impaired DNA repair capacity, and the onset of numerous age-associated pathologies

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*Correspondence: Chao Liang, [email protected] These authors have contributed equally to this work This article was submitted to Autoimmune and Autoinflammatory Disorders, a section of the journal Frontiers in Immunology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers