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To evaluate the cell migration, a transwell assay was conducted
Coagulation dysfunction is prevalent during sepsis, with the pathogenesis primarily involving the release of numerous inflammatory mediators, endothelial cell injury, activation of the extrinsic coagulation pathway, and suppression of anticoagulant and fibrinolytic systems (129)
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This observation aligns with recent studies on CRMP-2 and CRMP-5, which also identified these proteins in the nucleus 50,51
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