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Ethnic differences in association of outcomes with trimethylamine N-oxide in acute heart failure patients

As expected, we proved that boosting FAO via LCA, a natural and biologically active micronutrient enhancing physiological FAO through CPT1B (Marcovina et al., 2013) and activation of AMPK signaling pathway (Figures 3, 6), can significantly reduce obesity-mediated AF propensity and the corresponding atrial remodeling (Figures 1, 2), thus better supports the efficiency of FAO promotion in the AF therapeutic approach

Recent regulatory reviews by the MHRA and European Medicines Agency have assessed reports of psychiatric adverse events with GLP-1 receptor agonists, but no causal link has been established