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"I think it really is important now more than ever that we figure out a way to do a better job as physicians to check in on things like side effects, on things like muscle loss, on things like, 'how are your lifestyle changes going?' to make sure that our patients are safe," Guevarra said

Frataxin-knockout mouse myoblasts, heart, and skin fibroblasts all showed characteristics of ferroptosis, and Nrf2 was downregulated in Friedreichs Ataxia patients and frataxin-knockout mice, while Nrf2 activators EPI-743 or sulforaphane regulated redox imbalances and rescued ferroptosis in frataxin knockout cells

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doi: 10.1186/s12931-025-03356-w 23 HoggJCTimensW