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In cancer cells, increased lactate concentrations have been reported to suppress apoptosis through HIF-1-mediated upregulation of anti-apoptotic proteins, including BCL-2 and survivin, while concurrently promoting ferroptosis resistance via SCD1 upregulation (58,141)

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The biological rationale is that the body's healing program is itself coordinated, so supporting cell migration and local repair signaling together, rather than in isolation, is intended to better match how tissue actually rebuilds