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Glutamate and depression On the surface, this would appear to be a case of hypofunction or too little glutamate

A comparison with nonselective adrenergic agonists highlights albuterols selectivity, relative resistance to catechol-O-methyltransferase (COMT) metabolism, and primary use in acute asthma rescue therapy

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This broad spectrum of manifestations reflects the complexity of its pathophysiology, which involves a combination of dopaminergic neuron loss, oxidative stress, alpha-synuclein aggregation into Lewy bodies, mitochondrial dysfunction, neuroinflammation, and disruptions in the gut-brain axis 5,6,7,8,9