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Oxidative stress can lead to liver cell damage, inflammation, and fibrosis progression, while GSH can alleviate these effects by supplementing intracellular antioxidant reserves and stabilizing mitochondrial function [29, 30]
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We speculate that the BPC157-FBXO22 interaction via P3 delays enzymatic degradationpeptide catabolism typically initiates with endopeptidases, followed by stepwise proteolysis into amino acids
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