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Its mechanism centers on copper-dependent signaling that modulates gene expression across multiple tissue-repair pathways

However, when given subcutaneously, GHK-Cu bypasses this barrier and the peptide gets into circulation systemically at the desired dose

Polymers 15(4):901

Other researchers (252, 253) showed that the application of MDDP solution (pentoxifylline, glycine, deferoxamine, N-acetylcysteine, erythropoietin, MEL, and simvastatin) prior to liver reperfusion with HTK led to inhibition of malondialdehyde and IL-1 production, thereby completely abolishing the inflammatory response, reducing hepatocyte dysfunction and damage, and infiltration of the liver with inflammatory cells